cytotoxicity of icd-85 nps on human cervical carcinoma hela cells through caspase-8 mediated pathway

نویسندگان

saeed moradhaseli department of biochemistry, faculty of basic sciences, payame noor university, tehran, iran.

abbas mirakabadi department of venomous animals and antivenom production, razi vaccine and serum research institute, karaj, iran.

ali sarzaeem department of venomous animals and antivenom production, razi vaccine and serum research institute, karaj, iran.

morteza kamalzadeh department of quality control, razi vaccine and serum research institute, karaj, iran.

چکیده

the biological application of nanoparticles (nps) is a rapidly developing area of nanotechnology that raises new possibilities in the treatment of human cancers. the cytotoxicity was evaluated by mtt and ldh assays. the apoptotic effect of free icd-85 and icd-85 nps on hela cells was assessed using caspase-8 colorimetric assay. the mtt assay showed that icd-85 nps could enhance the in-vitro cytotoxicity against hela cellscompared to the free icd-85. the ic50 value at 72 h was reduced from 25 ± 2.9 μg/ml for free icd-85 to 15.5 ± 2.4 μg/ml for icd-85 nps. however, ldh assay demonstrated that icd-85 has dose-dependent cytotoxicity on hela cells while icd-85 nps exhibited weaker cytotoxicity on same cells. the results also indicate that icd-85-induced apoptosis on hela cells is associated with the activation of caspase-8. moreover, caspase-8 assay analysis demonstrated that the icd-85 nps induced a higher apoptotic rate in hela cells compared to free icd-85. our results demonstrated that the encapsulation of icd-85 enhances its anti-proliferative effects. taken together, these results suggest that the delivery of icd-85 in nanoparticles may be a promising approach for the treatment of the cancer.

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عنوان ژورنال:
the iranian journal of pharmaceutical research

جلد ۱۲، شماره ۱، صفحات ۱۵۵-۱۶۳

کلمات کلیدی
[ ' c a n c e r ' , ' n a n o p a r t i c l e s ' , ' i c d ' , 8 5 , ' h e l a c e l l l i n e ' , ' m t t a s s a y ' , ' c a s p a s e ' , 8 ]

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